One-dimensional modelling of the vulnerability to re-entry of homogeneous atrial tissue

H. Zhang; A. V. Holden

doi:10.1006/jtbi.1996.0280

One-dimensional modelling of the vulnerability to re-entry of homogeneous atrial tissue

H. Zhang, A. V. Holden

Research output: Contribution to journal › Article › peer-review

Abstract

A one-dimensional excitable medium model of atrial tissue derived from the Earm-Hilgemann-Noble excitation equations for atrial excitation is used to investigate (1) the rate dependence of the conduction velocity of travelling wave solutions and (2) vulnerability to unidirectional conduction block and hence re-entry. The effects of changes in the ACh activated K+ current and Na+/K+ pump inhibition on these properties are computed.

Original language	English
Pages (from-to)	117-122
Number of pages	5
Journal	Journal of Theoretical Biology
Volume	184
Issue number	2
DOIs	https://doi.org/10.1006/jtbi.1996.0280
Publication status	Published - 21 Jan 1997

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10.1006/jtbi.1996.0280

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title = "One-dimensional modelling of the vulnerability to re-entry of homogeneous atrial tissue",

abstract = "A one-dimensional excitable medium model of atrial tissue derived from the Earm-Hilgemann-Noble excitation equations for atrial excitation is used to investigate (1) the rate dependence of the conduction velocity of travelling wave solutions and (2) vulnerability to unidirectional conduction block and hence re-entry. The effects of changes in the ACh activated K+ current and Na+/K+ pump inhibition on these properties are computed.",

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AB - A one-dimensional excitable medium model of atrial tissue derived from the Earm-Hilgemann-Noble excitation equations for atrial excitation is used to investigate (1) the rate dependence of the conduction velocity of travelling wave solutions and (2) vulnerability to unidirectional conduction block and hence re-entry. The effects of changes in the ACh activated K+ current and Na+/K+ pump inhibition on these properties are computed.

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JF - Journal of Theoretical Biology

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One-dimensional modelling of the vulnerability to re-entry of homogeneous atrial tissue

Abstract

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